Neuroprotection in Brain Ischemia: An Update

Description

Despite the significant advances that have been made over the past decade in delineating the cellular mechanisms of ischemic brain injury, researchers have not been able to translate these gains in basic research into clinically effective neuroprotective treatments. One major line of investigation has involved the theory of excitotoxicity, which is based on the observation that large concentrations of glutamate can destroy neurons. Now investigators have identified new, glutamate-independent targets for neuroprotection in brain ischemia. The findings may have important implications for the development of a new clinical paradigm for neuroprotection, according to Roger P. Simon, MD. Dr. Simon is Director of Neurobiology Research and Chair in Neurology at the Robert S. Dow Neurobiology Laboratories in Portland, Oregon.

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